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Spinal canal stenosis, also referred to as spinal narrowing, represents a pathological condition characterized by structural abnormalities within the bony components of the vertebral column. These alterations result in a diminished available space inside the spinal canal, consequently leading to compression of the spinal cord and nerve roots, which in turn provokes a spectrum of clinical symptoms of varying severity. Although this condition may theoretically develop in any segment of the spine, it is most frequently observed in the cervical and lumbar regions—areas subjected to the highest biomechanical stresses—whereas thoracic spinal stenosis remains relatively uncommon. Epidemiological data indicate that the disorder affects both sexes with comparable frequency, with clinical manifestations typically becoming more pronounced after the age of 50. The etiology of spinal canal stenosis may be either congenital or acquired. Inborn anatomical anomalies do not necessarily present symptoms immediately at birth; instead, their clinical expression often emerges later in life, with external factors such as chronic postural defects or excessive static-dynamic loading accelerating their progression. Far more prevalent, however, are secondary stenoses arising from specific pathological entities. The predominant cause in clinically confirmed cases remains degenerative spinal disease, which facilitates the formation of osteophytes—bony outgrowths that exert pressure on neural structures. Additional predisposing factors include: excessive growth hormone secretion by the pituitary gland (acromegaly), Paget’s disease marked by disordered bone metabolism, intervertebral disc herniations causing displacement of the nucleus pulposus, pathological hypertrophy of spinal ligaments, mechanical trauma of varying severity, postoperative complications following spinal surgeries, ankylosing spondylitis, and other degenerative processes generating bony deformities that compress the spinal cord.

Spinal canal stenosis, a condition characterized by an irregular and highly variable clinical presentation, manifests differently in each patient depending on the location of pathological narrowing and individual physiological responses. Given that stenosis can occur in any of the three primary spinal regions—cervical, thoracic, or lumbar—each variant produces a distinct constellation of symptoms. In the cervical spine, patients frequently experience severe neck and occipital pain, often accompanied by episodes of vertigo and neurological deficits such as muscle weakness, sensory disturbances, or radiating pain into the upper extremities, which may be misdiagnosed as cervical radiculopathy. Lumbar stenosis, by contrast, typically presents with radicular pain that extends along the lower limbs, a pattern sometimes mistaken for sciatica. Progressive worsening of pain may lead to paresthesias—uncomfortable sensations of tingling or prickling—as well as gait disturbances and the adoption of compensatory, pathological movement patterns. In advanced cases, symptoms of cauda equina syndrome may emerge, including urinary and bowel dysfunction alongside significant lower extremity muscle weakness. The least common but clinically relevant thoracic stenosis often causes pain radiating to the ribs, scapulae, or occasionally the lower limbs, and in some instances, may even provoke pain during respiration.

The accurate identification of spinal stenosis—much like other neurological disorders—demands a meticulous diagnostic approach. The initial step involves a comprehensive neurological examination, supplemented by a thorough patient history, which facilitates the preliminary localization of pathological changes within the spine. To confirm the diagnosis with certainty, however, advanced imaging studies such as X-ray radiography (RTG) or magnetic resonance imaging (MRI) must be employed. Among these, MRI offers significantly greater precision, as it not only visualizes bony structures (including potential osteophytes) but also soft tissues such as spinal nerves—a level of detail unattainable through X-ray alone.