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Insufficiency of the thyroid gland

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Insufficiency of the thyroid gland

Of all thyroid disorders, this is the most common -- it is much more common in the elderly, but it's the greatest risk for pregnant women because it can lead to many complications -- find out if you can suspect thyroid dysfunction and how to diagnose it.

Table of Contents

1. What's the hypothalamus?

Thyroid insufficiency is a clinical condition caused by insufficient levels of thyroid hormones. The thyroid mainly produces thyroxine (T4) and triglyceride (T3). T3 is the more active version and is mainly produced from T4. The insufference may be primary or secondary. The primary cause of thyroxy inefficiencies is improper storage of the thyroid itself, its deficiency or damage. The secondary cause is TSH, i.e. a neurodegenerative condition that reaches thyroid receptors and causes it to work. The most common cause of the primary cause is the absence of a thyroid hormone. In order to assess the effectiveness of the antihypertensive effects of this disease, it is necessary to evaluate the presence of an antihistamine in thyroid tissue.

2. What are the symptoms of hypothyroidism?

The main symptoms of thyroid insufficiency include: a decrease in the rate of basic metabolism leading to an increase in body weight, reduced body temperature, impairment of concentration and memory, weakening of muscle strength, difficulty breathing due to weakness of the upper respiratory muscles, increased LDL cholesterol, anemia, constipation, dry, cold skin, crippled voice, loss of hair, yellowish-orange discoloration of the skin due to disorders of the cartilage to the aorta, increases in the cholesterol levels of men and women.

3. Diagnosis of the disease

Additionally, testing for TSH, FT4 (free thyroxine) and FT3 (free triiodothyronine) levels is essential in the diagnosis of thyroid insufficiency. FT4 levels are lowered, FT3 is normally normal. In primary incidence, usually an elevated level of TSH is observed, in secondary incidence is lowered. In addition, the test for anti-TPO (anti-hypertensive to hyperthyroidism) can be used to determine whether Hashimoto' s disease is involved.

4. Treatment of the inattention

Treatment of clear thyroid insufficiency in most cases is based on the lifelong use of synthetic levothyroxine. It is most commonly taken in the morning fasting, at least 30 minutes before the first meal.

5. What the menu should look like in the hypothalamus

It should be constructed according to the principles of rational nutrition, with particular emphasis on providing food with adequate doses of iron, vitamin B12, zinc and selenium. The current intake of iron and vitamin B12 from the digestive system should always be taken into account before the effective treatment of intestinal dysfunction can be initiated. Therefore, it is very important that we provide adequate supplies of these ingredients in a rational way. It is therefore important to ensure that we maintain adequate levels of protein in our dietary systems, so that the primary sources of energy in the dietary system can be converted into dietary supplements, otherwise it is essential to maintain a healthy diet and nutritional balance.

6. Physical activity and hypothyroidism

Regular physical activity can improve thyroid function. This is confirmed by a study conducted in India a few years ago in which 20 patients were treated for thyroid insufficiency. 10 of them were physically active (minimum of 1 hour per day). After 3 months in the exercise group, there was a significant decrease in TSH levels and an increase in T3 and T4 secretions, indicating that the thyroid was working better without increasing the dose. However, there were no changes in the control group.
Source

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Bansal A. et al., The effect of regular physical exercise on the thyroid function of treated hypothyroid patients: An interventional study at a tertiary care center in Bastar region of India, „Archives of Medical Science” 2015, 3, 244–246.
Sultan S., Rashed L., Effect of low kalorie diet and excercise on thyroid hormones and lepton levels, „The Medical Journal of Cairo University” 2009, 77(1), 33–39.
Thor P., Podstawy patofizjologii człowieka, Kraków 2009.